tous deposit along the coronary arteries, but not to such an extent as to reduce their calibre. The final cause of the stoppage of the heart was here found on the right side. The chordæ tendineæ of the tricuspid valve were looped together by filaments of fibrine, so that the curtains of the tricuspid were brought into approximation, and their action suspended. I have described this form of concretion in my work on Fibrinous Deposition in the Heart, and have there given a drawing, showing the way in which fibrinous bands may loop up the curtains of the tricuspid, which corresponds so absolutely with the appearances observed in Dr. Marshall's patient, that I reproduce it in fig. 1 of pl. II.

It is necessary, therefore, in examining the heart and blood-vessels in cases of cardiac apnoea, to remember the existence of these filamentous cords, and to look for them with delicate manipulation, as the mechanical obstacles which may have led ultimately to the arrest of the circulation.

The next pathological condition, most frequent after those conditions which have been described, consists in the presence of fibrinous deposition in the cavities of a heart which in other respects is healthy. We find the concretion, giving rise to cardiac apnoea, in a large number of cases of an inflammatory kind; in croup, for example. Here the concretion is usually met with on the right side of the heart, where it may fill the auricle, or the auricle and ventricle, and send a prolongation into the pulmonary artery. Such an extension of concretion is well depicted in fig. 2, pl.